Quote:
Originally Posted by Riot
Lower airway inflammation that affects the integrity of the alveolar-capillary interface and constriction of bronchioles (that can be contributory to EIPH caused by pressure difference) is different from upper airway inflammation, and organic and inorganic matter are rarely, if at all, associated with lower airway inflammation.
Bacteria and viruses can cause infection with subsequent scarring if they are respiratory pathogens and are inhaled, not removed by immunologic and physical defense systems, and set up housekeeping within the lungs. That takes at least 6 hours or longer, thus inhalation during a race is not contributory to EIPH that occurs during that race.
A previous episode of lung infection/pneumonia, if it causes scarring or lung damage, can in the future make the horse more susceptible to dorso-caudal lung lobe problems, however that is not the most common location within the lung of infection/pneumonia.
Medicine: it takes a thorough in-depth knowledge of normal anatomy and physiology before one can start identifying and speculating upon the abnormal.
Do you have a point, or are you just dancing for Freddy?
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As expected, you masterfully backpedaled from your earlier absolute that inhalants from racing don't affect EIPH. Antitrust had a good point that perhaps racing on dirt predisposes horses more to bleeding than on natural grass courses. No one said anything about bacteria, et al directly causing EIPH, nor suggested that inhaling kickback immediately led to EIPH in the same race. Only the notion that lower airway inflammation, for which many different microscopic sources found in dust/kickback are responsible, can exacerbate bleeding was offered. Seems like you agree even though your original response read the opposite way.
You're welcome.
05-14-2012, 09:58 PM
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